Autophagy and ubiquitin-proteasome system as potential mechanisms responsible for left ventricle mass loss in a monocrotaline pulmonary hypertension rat model.
2016/23/N/NZ5/00597
Keywords
Descriptors
- NZ4_4: Patofizjologia ogólna
- NZ5_9: Inne zagadnienia pokrewne
- NZ2_2: Genomika, transkryptomika i epigenomika
Panel
NZ5 – Human and animal noninfectious diseases: etiology, mechanisms, diagnosis and treatment of diseases, poisonings and injuries (without neurological diseases)
Host institution
Uniwersytet Jagielloński- Collegium Medicum, Collegium Medicum; Wydział Lekarski
Voivodeship: małopolskie
Principal investigator (from the host institution)
Number of co-investigators in the project: 2
Call
PRELUDIUM 12 – announced on 2016-09-15
- Amount awarded
- 150 000 PLN
- Project start date
- 2017-10-20
- Project end date
- 2020-10-19
- Project duration
- 36 months (the same as in the proposal)
- Project status
- Project settled
Project description
Download the project description in a pdf file
Note — project descriptions were prepared by the authors of the applications themselves and placed in the system in an unchanged form.
Information in the final report
Publication in academic press/journals (1)
-
Myocardial proteomic profile in pulmonary arterial hypertension
- Authors:
- Mateusz K. Hołda, Aneta Stachowicz, Maciej Suski, Dorota Wojtysiak, Natalia Sowińska, Zbigniew Arent, Natalia Palka, Piotr Podolec, Grzegorz Kopeć
- Academic press:
- Scientific Reports (year: 2020, volume: 10, pages: 16), Publisher: Springer Nature
- Status:
- Published
- DOI:
- 10.1038/s41598-020-71264-8 – link to the publication