Characterisation of leptin signalling in the obese uteri: putative link to transcriptional regulation during pre-decidualisation
2019/35/N/NZ4/03496
Keywords
Descriptors
- NZ2_002: Genomika, transkryptomika i epigenomika
- NZ4_006: Endokrynologia
- NZ4_011: Metabolizm
Panel
NZ4 – Biology of tissues, organs and organisms: morphology and functions of animal's and human's systems, organs and organisms, experimental medicine, basics of neurology
Host institution
Instytut Rozrodu Zwierząt i Badań Żywności PAN
Voivodeship: warmińsko-mazurskie
Principal investigator (from the host institution)
Number of co-investigators in the project: 3
Call
PRELUDIUM 18 – announced on 2019-09-16
- Amount awarded
- 140 000 PLN
- Project start date
- 2020-06-25
- Project end date
- 2023-06-24
- Project duration
- 36 months (the same as in the proposal)
- Project status
- Project settled
Project description
Download the project description in a pdf file
Note — project descriptions were prepared by the authors of the applications themselves and placed in the system in an unchanged form.
Information in the final report
Publication in academic press/journals (1)
-
Fetal growth restriction and placental defects in obese mice are associated with impaired decidualisation: the role of increased leptin signalling modulators SOCS3 and PTPN2
- Authors:
- Walewska, Edyta; Makowczenko, Karol G.; Witek, Krzysztof; Laniecka, Elżbieta; Molcan, Tomasz; Alvarez-Sanchez, Andrea; Kelsey, Gavin; Perez-Garcia, Vicente; Galvão, António M.
- Academic press:
- Cellular and Molecular Life Sciences (year: 2024, volume: 81, pages: art. no 329 (1-20)), Publisher: SPRINGER BASEL AG
- Status:
- Published
- DOI:
- 10.1007/s00018-024-05336-7 – link to the publication